Archive for category ‘Chemical Exposure‘

Export of waste electrical and electronic equipment (WEEE): plenty of gold, and toxic chemicals, too

Study shows need for action to promote ecological recycling

More than 155,000 tonnes of what is sometimes hazardous electronic waste are exported annually from Germany to non-European destinations, a volume which includes some 50,000 tonnes of PC and television monitors alone. The latter often contain metals as well as flame-retardant bromide compounds such as hazardous polybrominated diphenyl ether (PentaBDE). Even defective appliances are often re-classified as “functional”, then usually shipped to Asia and Africa where they are only rarely recycled ecologically. These are the findings of a new study commissioned by the Federal Environment Agency (UBA), which was presented at CEBIT in Hannover.

Federal Minister for Environment Dr. Norbert Röttgen said, “The study illustrates the scale of illegal export of WEEE while also pointing out that further measures to solve the problem are necessary. The Federal government is calling for a decisive regulation at the European level by which exporters must provide proof that exported devices are in working order and not in fact waste, and for exporters to bear the costs of periodic checks”. The appropriate authorities in Germany must step up monitoring of collection points and exports so as to curb the detrimental impact on environment and health of criminal trade.

“Not only do many hazardous materials leave the country in used electronic equipment, but valuable raw materials such as gold, copper, platinum or indium are also exiting the raw materials cycle here at home”, said UBA President Jochen Flasbarth. “As long as ecological recycling is technically and satisfactorily possible in Europe or similar regions only, equipment and its components should be reused here. It makes economic sense to recycle valuable raw materials appropriately, especially many metals, considering the rise in global market prices”, continued Flasbarth.

UBA’s President also made an appeal to continue improvement of recycling standards in Asia and Africa through technology transfer. Producers of new equipment are also called upon to design products more ecologically.

In their one-and-a-half-year-long study experts from the Hamburg Institute for Environmental Strategies (Ökopol) compiled the most solid information as yet on the origin and volume of exported devices. The equipment came from flea markets, second hand shops or were retrieved from junk yards. From there it is often transported via collection points for export, usually by sea. Besides harmless metallic raw materials, WEEE also houses a host of hazardous materials which must be recycled properly to avoid harming human health and the environment. An old computer contains more than 100 different materials, and conventional monitors contain lamps which must also be disposed of professionally.

The study findings are to be forwarded to affected stakeholders, in particular the concerned federal ministries, municipal umbrella organisations, environmental and economic associations, and the European Commission.

The “Optimierung der Steuerung und Kontrolle grenzüberschreitender Stoffströme bei Elektroaltgeräten/Elektroschrott” study (in German with English-language summary) is available as a free download at www.umweltbundesamt.de. A background paper is available at www.bmu.de (in German).

Literature: Umweltbundesamt, BMU – German Federal Ministry for Environment, Nature Conservation and Nuclear Safety, Export of waste electrical and electronic equipment (WEEE): plenty of gold, and poison, too, Press release No. 029/10, Berlin, 04.03.2010

Study finds dirty air in California causes millions worth of medical care each year

California’s dirty air caused more than $193 million in hospital-based medical care from 2005 to 2007 as people sought help for problems such as asthma and pneumonia that are triggered by elevated pollution levels, according to a new RAND Corporation study.

Researchers estimate that exposure to excessive levels of ozone and particulate pollution caused nearly 30,000 emergency room visits and hospital admissions over the study period. Public insurance programs were responsible for most of the costs, with Medicare and Medi- Cal covering more than two-thirds of the expenses, according to the report.

“California’s failure to meet air pollution standards causes a large amount of expensive hospital care,” said John Romley, lead author of the study and an economist at RAND, a nonprofit research organization. “The result is that insurance programs — both those run by the government and private payers — face higher costs because of California’s dirty air.”

While much work has been done previously to catalog the economic impact of air pollution across California, the RAND study is the first to quantify the cost of hospital-based medical care to various payers caused by the failure to meet federal clean air standards across the state. More people in California live in areas that do not meet federal clean air standards than in any other state.

Romley said the findings show that private insurers, employers and public insurance programs all have a financial stake in improving California’s air quality.

“These costs may not be the largest problem caused by dirty air, but our study provides more evidence about the impact that air pollution has on the state’s economy,” Romley said.

Researchers used records from air pollution agencies and hospitals to estimate how failing to meet federal and state standards for particulate matter and ozone would affect private and public insurer spending for hospital admissions for respiratory and cardiovascular causes, and emergency room visits for asthma throughout California from 2005-2007.

Researchers say the most common hospital-based medical care triggered by elevated air pollution levels are emergency room visits for asthma among children aged 17 and under, with more than 12,000 visits over the three-year study period.

The most costly conditions examined by researchers were hospital admissions triggered by air pollution for acute bronchitis, pneumonia and chronic obstructive pulmonary disease. Those conditions accounted for nearly one-third of the $193 million in health care spending documented over the study period.

Nearly three-quarters of the health events identified by researchers were triggered by high levels of fine particulate pollution — tiny pieces of soot that can lodge deep in lungs. The health events examined in the study were concentrated in the San Joaquin Valley and the four-county South Coast Air Basin.

The cost of treating health events caused by air pollution is equal to the expense of providing flu vaccines to 85 percent of California children under age 15, according to the report.

Researchers say their study provides a conservative estimate about the costs of medical care triggered by air pollution because it does not include outpatient care provided in clinics or medical offices. Details about that type of medical care are not routinely reported to state agencies and thus could not be analyzed.

The study also includes case studies of individual hospitals in Fresno, Lynwood, Palo Alto, Riverside and Sacramento. That analysis demonstrates that costs and types of illness reported vary by region.

To conduct the study, researchers used epidemiological studies that link elevated pollution levels to respiratory and cardiovascular illnesses, and compared that information to pollution levels measured across the state from 2005 to 2007 by various public agencies. Researchers also reviewed detailed records hospitals report to the state about the patients they treat, the illnesses diagnosed and who pays for that care.

Literature:

RAND Corporation*, Study finds dirty air in California causes millions worth of medical care each year, March 2, 2010

*The RAND Corporation is a nonprofit research organization providing objective analysis and effective solutions that address the challenges facing the public and private sectors around the world.

Yale: Why BPA leached from ’safe’ plastics may damage health of female offspring

Yale scientists show how bisphenol A induces epigenetic changes in pregnant mice that cause hormonal imbalance in the later life of female progeny

Here’s more evidence that “safe” plastics are not as safe as once presumed: New research published online in The FASEB Journal suggests that exposure to Bisphenol A (BPA) during pregnancy leads to epigenetic changes that may cause permanent reproduction problems for female offspring. BPA, a common component of plastics used to contain food, is a type of estrogen that is ubiquitous in the environment.

“Exposure to BPA may be harmful during pregnancy; this exposure may permanently affect the fetus,” said Hugh S. Taylor, Ph.D., co-author of the study from Yale University School of Medicine in New Haven, Connecticut. “We need to better identify the effects of environmental contaminants on not just crude measures such as birth defects, but also their effect in causing more subtle developmental errors.”

Taylor and colleagues made this discovery by exposing fetal mice to BPA during pregnancy and examining gene expression and DNA in the uteruses of female fetuses. Results showed that BPA exposure permanently affected the uterus by decreasing regulation of gene expression. These epigenetic changes caused the mice to over-respond to estrogen throughout adulthood, long after the BPA exposure. This suggests that early exposure to BPA genetically “programmed” the uterus to be hyper-responsive to estrogen. Extreme estrogen sensitivity can lead to fertility problems, advanced puberty, altered mammary development and reproductive function, as well as a variety of hormone-related cancers. BPA has been widely used in plastics and other materials. Examples include use in water bottles, baby bottles, epoxy resins used to coat food cans, and dental sealants.

“The BPA baby bottle scare may be only the tip of the iceberg.” said Gerald Weissmann, M.D., Editor-in-Chief of The FASEB Journal. “Remember how diethylstilbestrol (DES) caused birth defects and cancers in young women whose mothers were given such hormones during pregnancy. We’d better watch out for BPA, which seems to carry similar epigenetic risks across the generations. ”

Author: FASEB* – Federation of American Societies for Experimental Biology, Why BPA leached from ’safe’ plastics may damage health of female offspring, 25-Feb-2010.

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* FASEB comprises 23 societies with more than 90,000 members, making it the largest coalition of biomedical research associations in the United States

Diabetes – Bitter Sweet or Toxic?

Indigenous people, diabetes and the burden of pollution

Diabetes is now widely regarded as the 21st century epidemic. With some 284 million people currently diagnosed with the disease, it’s certainly no exaggeration-least of all for Indigenous people.

According to the State of the World’s Indigenous Peoples Report by the United Nations, more than 50 per cent of Indigenous adults over the age of 35 have Type 2 Diabetes, “and these numbers are predicted to rise.”

Diabetes is referred to as a “lifestyle disease,” its rampant spread believed to be caused by obesity due to our increased reliance on the western diet (also known as the “meat-sweet” diet) and our avoidance of regular exercise.

While these may certainly be contributing factors, there is growing evidence that diabetes is closely linked with our environment. More than a dozen studies have been published that show a connection between Persistent Organic Pollutants (POPs) including polychlorinated biphenyls (PCBs); carcinogenic hydrocarbons known as Dioxins; and the “violently deadly” synthetic pesticide, DDT and higher rates of the disease.

“If it is the POPs, not the obesity that causes diabetes, this is really striking if true,” says Dr. David O. Carpenter, director of the Institute for Health and the Environment at the University of Albany.

One out of four Indigenous adults living on reserves in Canada have been diagnosed with Type 2 Diabetes, the most common form of diabetes. The prevalence of the disease appears to be so great that the number of new cases being diagnosed in Canada may exceed the growth of the Indigenous population. It’s no longer uncommon to find children as young as three with the disease. According to government statistics, 27 per cent of all Indigenous people in Canada will have Type 2 Diabetes in the next ten years.

Sandy Lake First Nation, in the Sioux Lookout Zone of northern Ontario, has all but met the mark. A March 2009 study co-authored by Dr. Stewart Harris found that 26 per cent of the community has the disease, the highest recorded rate of diabetes in Canada. With a population of 2,500, the northern Cree community was recently described as an “epicentre” of the epidemic.

There has been little research on the levels of persistent organic pollutants in Sandy Lake; however, according to the First Nations Environmental Health Innovation Network, several neighboring communities who also have high rates of diabetes, like Kitchenuhmaykoosib Inninuwug First Nation, are known to have elevated levels of PCBs in their blood.

The Mohawk community of Akwesasne has its own conflict with diabetes and exposure to POPs. Located across the New York-Ontario-Quebec borders along the St. Lawrence River, three aluminum foundries upriver from the reserve dumped PCBs into the river for decades, contaminating the water, soil, and vegetation.

For many years, Dr. Carpenter has been involved in the study of Adult Mohawks at Akwesasne. Most recently, in 2007, he took part in a study to examine the diabetes/pollution link in the community.

“Our study of adult Mohawks showed a striking elevation in rates of diabetes in relation to blood levels of three persistent organic pollutants, DDE, the metabolite of DDT, hexachlorobenzene and PCBs,” Dr. Carpenter explains. “Our results are quite compatible with those of Lee et al.”

In 2006, Dr. Dae-Hee Lee and her colleagues showed that people with the highest rate of exposure to POPs were roughly 38 times more likely to have diabetes than those with the lowest rate of exposure. Further, “they showed that people who were obese but did not have high levels of POPs were not at increased risk of developing diabetes,” continues Dr. Carpenter. “Probably the reason most people get obese is that they eat too many animal fats, and this is where the POPs are.”

The dietary source of POPs was confirmed by the US Environmental Protection Agency in their Draft 1994 Dioxin Reassessment, which has never been formally released to the public. According to the Draft Reassessment, 93 per cent of our exposure to Dioxin comes from the consumption of beef, dairy, milk, chicken, pork, fish, and eggs; in other words, the western diet.

A May 2001 study published in the Journal of Toxicology and Environmental Health drew similar conclusions to the EPA Reassessment. In addition, the study found that “nursing infants have a far higher intake of dioxins relative to body weight than do all older age groups,” and that human breast milk was twice as toxic dairy milk. It also found that vegans had the overall lowest rate of POPs in their bodies.

According to an October 2009 paper by the Research Centre for Environmental Chemistry and Ecotoxicology at Masaryk University, another major source of POPs, specifically DDT, is the world’s oceans. The paper also found that despite restrictions placed on the use of DDT more than 30 years ago, concentrations of the toxin are on the rise.

Indigenous people carry an unequally high proportion of this global toxic burden. For instance, according to Environment Canada’s National Pollutant Release Inventory (NPRI) there are 212 Indigenous communities in Canada living near or downstream from pulp mills and other facilities that produce dioxins and furans. One striking example is the old Dryden pulp mill near Grassy Narrows which, according to the Grassy Narrows and Islington Bands Mercury Disability Board, dumped tonnes of dioxin-laced mercury wastewater into the English-Wabigoon River system from 1962-70.

Forty years later, the poisonous waste continues to pose a “serious health threat” to Grassy Narrows and the Wabaseemoong First Nations, says the Disability Board. No formal steps have been taken toward remediation by federal or provincial governments.

The Tohono O’odham Nation’s experience bears a close resemblance to Grassy Narrows: the world’s highest rate of diabetes can be found in the southwest Arizona nation. According to Tribal health officials, nearly 70 per cent of the population of 28,000 has been diagnosed with the illness. The O’odham People make up the second largest Indigenous Nation in the United States.

Lori Riddle is a member of Aquimel O’odham Community and founder of the Gila River Alliance for a Clean Environment (GRACE).

GRACE was instrumental in the 10 year struggle against a hazardous waste recycling plant that operated without full permits on O’odham land for decades. Owned by Romic Environmental Technologies Corporation, the plant continuously spewed effluents into the air until it was finally shut down in 2007.

The Romic plant was not the first contributor to the O’odham’s toxic burden, explained Riddle. Looking back to her childhood, she recalled: “For nearly a year, [when] a plane would go over our heads, you could see the mist. We never thought to cover our water. The chemicals just took over and they became a part of us.”

From the early 1950s until the late 60s, cotton farmers in the Gila River watershed routinely sprayed DDT onto their crops to protect them from bollworms. According to the Agency of Toxic Substances and Disease Registry (ATSDR), each and every year, the farmers used roughly Twenty-three pounds of DDT per acre.

In 1969, the State of Arizona banned the use of DDT; by this time the river was gravely contaminated. According to the ATSDR, farmers then switched to Toxaphene, a substitute for DDT-until it was banned by the US government in 1990.

Because of these chemicals, Riddle explains, the O’odham were forced to abandon their traditional foods and adopt a western diet. Farms also went into a recession, forcing many families to leave their communities. Companies, such as Romic, began moving on to their territory, exasperating the situation. “It’s taken a toll on our quality of life,” she says. “I’ve cried myself to sleep.”

The O’odham are dealing with what Riddle terms “cluster symptoms” including miscarriages, arthritis in the spine, breathing problems, unexplainable skin rashes, and problems regenerating blood cells. This in addition to diabetes, which frequently leads to renal failure, blindness, heart disease, and amputations.

More and more studies are being published that show the link between diabetes and persistent organic pollutants like DDT-stemming from the landmark “Ranch Hand” study. In 1998, the study found a 166 per cent increase in diabetes (requiring insulin control) in US Air Force personnel who were sprayed with the herbicide and defoliant Agent Orange during the Vietnam War. The study also found that as dioxin levels increased so did the presence and severity of Type 2 diabetes, the time to onset declined following a similar trend.

However, Dr. Carpenter notes that because of the widely-endorsed belief that diabetes is a life-style disease related to diet and exercise, the link is gaining little attention by governments, news agencies, or by any of the hundreds of non-profit diabetes foundations around the world. “[It] hasn’t even made it into the medical community at this point,” Dr. Carpenter adds. “It takes a long time to change both medical and public opinion.”

“Clearly one thing everyone can do is to eat less animal fats,” suggests Dr. Carpenter. Several Indigenous communities in northern Manitoba and British Columbia have begun to do this, planting their own gardens and building greenhouses; returning, in a traditional sense, to some of the foods that sustained them for millennia. Others are turning to exercise, which plays a vital role not just in the prevention of diabetes, but in their overall health.

“Also, we must find ways of getting the POPs out of the animals that we eat. That is not going to be easy, given how contaminated we have made the world,” adds Dr. Carpenter. For this, Lori Riddle, who is herself a diabetic, points to the Tribal Council and the Federal Government.

Author and Copyright:

John “Ahniwanika” Schertow is an Indigenous rights advocate and author of the blog, Intercontinental Cry. / Contact

METAMORPHOSIS INSIDE MULTIPLE CHEMICAL SENSITIVITY

During our lives we suffer several metamorphoses, some are painful, others are positive, chosen or not. The experience, the life itself, makes us change and evolve.

My story is not different, although my most radical metamorphosis was when I fell ill with Multiple Chemical Sensitivity. But although I got sick suddenly, the process itself happened slowly. I was preparing for MCS for many years before I was aware of it. My body was warning me repeatedly without my understanding what it wanted to tell me. But how could I know that everything happening to me was the prelude to MCS? It’s almost impossible to know since information about MCS is kept secret from the public and when anyone dares to raise a voice, they are automatically silenced by those who say MCS is all in the minds of the patients.

It’s not easy to understand what happens to you as you search for a diagnosis, all the while trying not to fail during the long journey while you are riddled with attempts to damage your self-esteem as you struggle with a more diminished health status every day. The last stage of this particular metamorphosis happens when you finally know what it is happening: you have MCS. And then you start to reconsider the life you have known before in order to adapt yourself and to survive into the future.

All of us have gone through the stage of crying over things that we have lost, to hate what we have become. Where is that tireless and impulsive person who took the world by storm? It’s a natural, healthy and necessary stage. But oddly, then comes the most difficult thing: to find our place in this new world in which we’re doomed to live.

And surprisingly, when I thought that my life couldn’t be more foreseeable and monotonous, from the prison that my house has become, another metamorphosis started, this time deeper and visceral. This time my metamorphosis was chosen.

The need to communicate, to let the world know that I’m still alive, to cry out for my own rights and the rights of millions of people who suffer MCS in the whole world, led me to write. My timid voice started to be heard on my blog, No Fun, and then gathered strength thanks to Delirio’s articles, which were translated into several languages. And the first of them, “The Naked Truth about MCS,” was read on the Spanish Radio 3 program Carne Cruda. It was then that I finally dared to do something I had never imagined I would ever do: to write a book.

The extremely reserved person that I used to be has disappeared, in order to be able to tell my story to the world, as I dig into the deepest places of my being. Missing: A Life Broken by Multiple Chemical Sensitivity is a fulfilled wish as I report the situation in which we live. It’s my metamorphosis inside the metamorphosis of living with MCS. It’s my testimony, my life, my reflections. It’s also my contribution to the fight we’re doing at an international level to have MCS fully recognized. My book is the clearest proof that MCS didn’t take away my essence or my attitude; MCS didn’t steal my dreams but rather it changed my dreams so that I could help others.

My wish is that a lot of books will be written by people who are “missing” because of MCS so that the public knows we exist. We are ill, but no one will silence us.

Author: Eva Caballé / No Fun Blog, published at Delirio 2010.

Translation: Oscar Varona (from Delirio’s team) and Eva Caballé with help from Susie Collins.

Japanese and German versions are following soon.

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